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Pneumonia is suspected in any patient who has
fever, cough, chest pain, shortness of breath, and increased
respirations (number of breaths per minute). Fever with a shaking
chill is even more suspicious. Many patients cough up clumps of
sputum, commonly known as spit. These secretions are produced in the
alveoli during an infection or other inflammatory condition. They
may appear streaked with pus or blood. Severe pneumonia results in
the signs of oxygen deprivation. This includes blue appearance of
the nail beds or lips (cyanosis).
The invading organism causes symptoms, in part,
by provoking an overly-strong immune response in the lungs. In other
words, the immune system, which should help fight off infections,
kicks into such high gear, that it damages the lung tissue and makes
it more susceptible to infection. The small blood vessels in the
lungs (capillaries) become leaky, and protein-rich fluid seeps into
the alveoli. This results in less functional area for oxygen-carbon
dioxide exchange. The patient becomes relatively oxygen deprived,
while retaining potentially damaging carbon dioxide. The patient
breathes faster and faster, in an effort to bring in more oxygen and
blow off more carbon dioxide.
Mucus production is increased, and the leaky
capillaries may tinge the mucus with blood. Mucus plugs actually
further decrease the efficiency of gas exchange in the lung. The
alveoli fill further with fluid and debris from the large number of
white blood cells being produced to fight the infection.
Consolidation, a feature of bacterial pneumonias,
occurs when the alveoli, which are normally hollow air spaces within
the lung, instead become solid, due to quantities of fluid and
debris.
Viral pneumonias and mycoplasma pneumonias, do
not result in consolidation. These types of pneumonia primarily
infect the walls of the alveoli and the parenchyma of the lung. |